Panel Interviews

HEART FAILURE IN ADULTS

Professor Dr. Bilal Zikriya Khan
MBBS, MRCP(UK),MRCP(IRE)FRCP(Edin)

Professor of Cardiology
King Edward Medical College,
Head of Mayo Institute of Cardiology, Mayo Hospital, Lahore.

Consultant Cardiologist
Ammar Medical Complex
Tel no 5754916-19

103-B
GOR III
Shadman Lahore

 


Grading of Heart Failure: How much activity is permitted by heart failure!

I = Patient can perform routine activities without any discomfort.
II = Patient feels mild discomfort in performing routine activities and this slightly restricts his daily activities.
III = There is moderate to severe restriction of routine daily activities due to breathlessness. At rest he is without any discomfort.
IV = Patient is breathless even at rest.

(These are New York Heart Association guidelines)

 

NYHA classification of heart failure


Class I: asymptomatic
No limitation in physical activity despite presence of heart disease. This can be suspected only if there is a history of heart disease which is confirmed by investigationsfor example, echocardiography

Class II: mild
Slight limitation in physical activity. More strenuous activity causes shortness of breath for example, walking on steep inclines and several flights of steps. Patients in this group can continue to have an almost normal lifestyle and employment

Class III: moderate
More marked limitation of activity which interferes with work. Walking on the flat produces symptoms

Class IV: severe
Unable to carry out any physical activity without symptoms. Patients are breathless at rest and mostly housebound



Types of Heart Failure

Many Parameters

A Systolic or Forward Heart Failure: Basically enough pressure is not being created by myocardium to propel blood in systemic circulation. Myocardial function is compromised due to any reason. Infarction is one example.

Diastolic Heart Failure: Myocardium is stiff, cannot dilate. There can be multiple reasons, like amyloid deposition, fibrosis etcetera are many cardiomyopathies in this etiology. Filling of heart is restricted. This reduces force of contraction. Fourth heart sound is audible. No third heart sound is usually present. Murmurs are more common. Eiosinophilia may be associated with many myopathies.

B High Output Failure: Circulating blood volume is increased for prolonged periods of time. Heart cannot maintain adequate pressure due to increased load. Thyrotoxicosis, Anaemia & Beri Beri is few examples.

Low Output Failure:

C Hypertrophy Heart Failure: There are many inherited myopathies where myocardium is hypertrophied and it impairs filling which leads to failure.

D Right Heart Failure/Left Heart Failure.
Left heart failure eventually leads to right heart failure. Therapy remains the same.
 

 

Right Heart Failure

 

 

Left Heart Failure

  • Breathlessness
  • Breathlessness
  • Swelling of face or limbs or trunk.
  • Murmurs may be present
  • Ascites
  • Pulmonary congestion
  • Engorged Neck Veins.
  • Gallop Rhythm



Symptoms and Signs

Symptoms

Following are the main symptoms.

*
* Breathlessness
* Oedema, Swelling.
* Weakness
* Lethargy
* Tachycardia
* Palpitations
* Giddiness
* Symptoms of Primary diseases.
*

There is a long list of other symptoms. Whenever heart failure is present above mentioned first two symptoms are usually present even when patient presents with some other complaint.

Breathlessness

A long list of causes is present. At times difficulty arises to differentiate the origin of this symptom. Is this due o cardiac failure or due to Lung disease? Following features should be elicited by cross-questioning.


 

Breathlessness due to cardiac origin

Breathlessness due to lung disease

Most important feature is relation to exertion. Severity of symptom is related to degree of exertion.

Here important feature is level of bronchospasm which is related to

  1. Seasonal variation
  2. Diurnal Variation
  3. Occupation

Problem persists for most of the times.

Even if it is continuous now, it used to be episodic in the past.

Cough is not productive

Cough may be productive

Patient feels better while lying down

Patient feels better sitting up.

There is usually no wheezing.

Wheezing may be audible. Whistles.



Signs of Heart Failure

Swelling, edema or bloated ness
Tachycardia
Gallop Rhythm
Wheezing or crackles may be present.
Heart Murmurs may be present
Engorged Neck Veins.




Investigations

Impression of Prof Bilal Zikerya is that delay in early detection of heart failure patients is due to delay in ordering two basic investigations.

* X-ray Chest
* ECG

For early detection all patients who raise little bit of suspicion of heart failure, where physician has the impression that breathlessness may be of cardiac origin, and there are risk factors predisposing to heart failure, these two investigations should be ordered immediately.

X-ray Chest

In the present of normal size of heart: Features of left ventricular failure should be looked for. These are pulmonary congestion, typical batwing shadowing or upper lobe congestion.
Where size of heart is enlarged: Other features of heart failure should be searched for.

ECG

All patients who are suspected of heart failure or who have any risk factor should have ECG done at earliest. Signs of ischaemia, heart enlargement or any silent infarction may be picked up.

Patients who are diagnosed as patients of heart failure on history, examination, Xray chest and ECG should have following baseline investigations.

* Urine: Routine Analysis.
* Sugar: Blood Levels.
* Lipid profile
* Renal Profile: Serum Creatinine and blood urea in addition to electrolytes. Whenever therapy is started for heart failure, it is preferable to repeat the renal profile after 2 weeks to estimate any disturbance in renal function being caused by diuretics or ACE inhibitors.
* CBC: Anaemia is quite common and it is being overlooked.


If response to therapy is not as expected then

* ETT
* Echocardiography
* Pulmonary Function Tests

Should be ordered to further define the cause of symptoms.
Echocardiography can be ordered to quantify heart failure and to look into the causes of heart failure even when diagnosis of heart failure is established without this investigation. Diagnosis of heart failure can be established without echocardiography.


Common Risk Factors for Heart Failure

* Hypertension
* Ischaemic Heart Disease
* Valvular Heart Disease
* Cardiomyopathies
* Rheumatic Heart Disease


Evaluation of Heart Failure

* First of all remediable causes, like Valvular heart disease, should be ruled out. If any is present then surgery should be advised at appropriate time without delay.

* Look for Ischaemic Heart Disease.30-40% infarctions may be silent. I H D should be properly evaluated by proper persons to judge whether any by-pass surgery can be beneficial or not.

* A functional grading should be done according to NYHA criteria. This should help to monitor and evaluate therapy and measure the prognosis.

* All etiologies which can be of primary importance or detrimental in nature like Diabetes, Hypertension or renal failure should be properly assessed and managed.

Therapeutic Tools

Diuretics

Following diuretics can be used according to the amount of diuresis needed.

Loop Diuretic: Frusamide; Most powerful diuretic. Potassium supplements should be routinely added. If BP is below 90/60 initially in heart failure then it is better to admit the patient and give the diuretics in combination with inotropic drugs like dobutamide. Frusamide can be combined with potassium sparing diuretics.

Thiazides: These are less powerful. Loss of potassium is less. So where diuresis is intended to be mild these can be used instead of frusamide.

Spironolactone: There is now increased interest in this diuretic. Diuretic effect is very mild but this has the capacity to delay or minimize fibrosis in myocardium.


ACE Inhibitors

These should be used in all kinds of heart failure. Wherever there is major metabolic disturbance like in Diabetes, these are more beneficial. In diabetics renal profile can be repeated after 2 weeks of use to assess any disturbance caused by ACE Inhibitors.

ARBs like Losartan

These can be used in place of ACE inhibitors where tolerance is a problem with the liters due to cough. Equally effective.

Betablockers

These should be used in all patients of heart failure where there is sympathetic overstimulation. Resting Tachycardia is one such indication. These should not be used in Valvular heart disease. Care should be observed in patients of systolic dysfunction. Beta-blockers should be titrated very slowly, starting with slowest possible dose. Carvedilol (Dimitone) is probably the best studied for this use. Metoprolol is another alternative.

Nitrates, Vasodilators, Calcium Chanel Blockers

These can be used in heart failure. Mild beneficial effects are there. CCBs are not much effective. Where ACE inhibitors cannot be used due to cough then one of these can be added.

Digoxin

Current opinion in the west is that Digoxin should be used only in those patients of heart failure where atrial fibrillation is also present. In Pakistan due to poor economic conditions it should be used on wider scale.

In Emergencies

If heart failure is ascertained then Diuretic and Oxygen should be started immediately pending other investigations. But if diagnosis is in doubt and patient is in acute distress then it is better to nebulize the patient, give oxygen and start investigating patient immediately.

In Pregnancy

ACE inhibitors are absolutely contraindicated. Nitrates can be used at any stage. Diuretics have to be used if acute heart failure is there and gyaenocologists suggest prolonging the gestation.

Few terms about the Myocardium
In the background of I H D & Heart Failure

Remodeling: After infarction myocardium reorganizes itself. There has to be fibrosis.Remodelling refers to this reorganisation.Spironolactone and ACE inhibitors are known to have beneficial effects in this regard.Vasodilators like amlodipine and nitrates may also be useful.

Hibernatin: After AMI when remaining cardiac function is correlated with the loss of myocardial tissue, sometimes it is seen that function is much more disturbed then the estimated loss of myocardium suggests. Angiography and other investigations suggest certain areas where blood supply is not completely lost but it is reduced to the levels where myocardium is alive but has gone to low metabolic state due to ischaemia. Function of these areas is not appropriate. These areas are said to have gone into hibernation. Effective therapeutic measures can be taken to bring these areas to full function.

Stunning: It refers to similar state of affairs, as in hibernation in the adjoining areas of infarction. Here function is impaired due to Ischaemic but cells do not die.

Processes of remodelling, hibernation, and stunning take months to stabilize.